Cloze Splitter – Automatically Split Multi-Cloze Notes into Separate Cards

AnkiWeb addon 424558481

Automatically detects cloze deletions and splits entries separated by | into separate cards, each containing only its selected cloze while preserving original cloze numbers.
AI-generated summary; may contain mistakes.

card-creationnote-managementcloze

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Votes
0 (👍 0 · 👎 0)
Updated
2025-12-02
Anki versions
2.1.1~
License
Not found (this does not mean that the addon has no license)
Virus scan
No ClamAV match A match does not prove malware. No match does not prove that the code is safe.
Languages
Python
Description language
en

Maintenance

stale

  • Last update or commit: 2025-12-02 (334 days before the snapshot).

GitHub

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Version branches
Min AnkiMax AnkiUpdated
2.1.1+2025-12-02
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Description

Automatically detect all cloze deletions { {c1:: ... } }, { {c2:: ... } }, etc. Split them into separate notes, each containing only the selected cloze.

Usage:

*{{c1::C5a>СЗа>С4а::С5а/С3а/C3b}} = anaphylatoxins (histamine release → vascular permeability)|

  • {{c2:: 5а:С5а/С3а/C3b}} = strongest chemotactic factor for neutrophils|
  • {{c2:: C3b::C5a/C3a/C3b}} = opsonisation

Separated by "|", it will generate 3 cards.

Note: Though origial text contains one C1 and two C2, it will be converted to C1 first. Therefore, acturally, it will generate 3 cards!

The idea is from https://www.reddit.com/r/Anki/comments/1oko6rz/comment/nolbzr8/?context=1 .

Change 2025-12-02 Stop converting all Cloze numbers to C1

For example:

{{c1::Citrin}} = Aspartate–glutamate mitochondrial transporter (exports {{c2::aspartate to cytosol}}). Defect → Aspartate trapped in {{c3::mitochondria}} → cytosolic aspartate deficiency| {{c1::Citrulline}} cannot combine with aspartate → no {{c2::argininosuccinate}} formation → urea cycle stops| {{c1::Malate}}–aspartate shuttle impaired → ↑NADH cytosol → carbohydrate intake worsens symptoms. Result → {{c2::hyperammonemia + neurotoxicity}}.

This should be converted to 3 different cards

{{c1::Citrin}} = Aspartate–glutamate mitochondrial transporter (exports {{c2::aspartate to cytosol}}). Defect → Aspartate trapped in {{c3::mitochondria}} → {{c3::cytosolic aspartate}} deficiency

{{c1::Citrulline}} cannot combine with aspartate → no {{c2::argininosuccinate}} formation → urea cycle stops

{{c1::Malate}}–aspartate shuttle impaired → ↑NADH cytosol → carbohydrate intake worsens symptoms. Result → {{c2::hyperammonemia + neurotoxicity}}.